![]() It consists of the following three pathophysiologic processes: “ Acute asthma exacerbations” and “ Exercise-induced bronchoconstriction” are discussed in their own articles.Īsthma is an inflammatory disease driven by T-helper type 2 cells ( Th2-cell) that manifests in individuals with a genetic predisposition. Follow-ups are essential to monitor the response to therapy and to adjust treatment regimens in a stepwise manner. To achieve symptomatic control and minimize the risk of exacerbations, comorbidities should be managed and exposure to asthma triggers minimized. ![]() Systemic corticosteroids are usually reserved for patients with severe persistent asthma. Treatment regimens differ based on the severity of asthma but primarily consist of different combinations of beta-2 agonists and inhaled corticosteroids ( ICS). Additional tests may be required to evaluate for asthma triggers and comorbidities that increase the risk of acute exacerbations. In a patient with typical clinical features of asthma, diagnosis is confirmed by demonstrating reversible bronchial obstruction on pulmonary function tests. Symptoms remit in response to antiasthmatic medications or resolve spontaneously upon removal of the trigger. ![]() The cardinal symptoms of asthma are intermittent dyspnea, cough, and high-pitched expiratory wheeze. Nonallergic (environmental or intrinsic) asthma usually develops in patients over the age of forty and can have various triggers, such as cold air, medications (e.g., aspirin), exercise, and viral infections. Allergic (extrinsic) asthma usually develops in childhood and is triggered by allergens such as pollen, dust mites, and certain foods. Asthma is a chronic inflammatory disease of the respiratory system characterized by bronchial hyperresponsiveness, episodic acute asthma exacerbations, and reversible airflow obstruction.
0 Comments
Leave a Reply. |
AuthorWrite something about yourself. No need to be fancy, just an overview. ArchivesCategories |